Superior cerebellar artery

Every tissue structure in the human brain is very important, so in order to avoid the occurrence of diseases, everyone must protect their brain. Blood in the brain is mainly supplied by arteries, so there are arteries distributed in basically every part of the brain. The cerebellum is also a very important tissue structure in the human brain, but what happens when cerebellar artery occlusion occurs?

Occlusion of the superior cerebellar artery can cause three clinical syndromes in the cerebellum, pons, and midbrain:

(1) The pons may present with the lateral superior pontine syndrome, also known as the pontine tegmental syndrome or Raymond-Cesten syndrome, which manifests as: ① pain and temperature sensation impairment on the contralateral half of the body (including the face) (damage to the trigeminal lemniscus and spinothalamic tract); ② ipsilateral chewing

Muscle paralysis (involvement of the motor nucleus of the trigeminal nerve); ③ Ipsilateral Homer sign (damage to the descending sympathetic fibers of the reticular formation); ④ Ipsilateral limb cerebellar ataxia (combined arm damage). In addition, if the central bundle of the tegmentum is damaged, limb muscle clonus may occur; medial

Nystagmus may occur when the longitudinal fasciculus is damaged.

(2) The midbrain may produce tegmental syndrome, which is manifested as: ① cerebellar ataxia, poor coordination and decreased muscle tone of the ipsilateral limbs; ② tremors and chorea-like movements. The short circumflex artery that supplies the sole of the cerebral foot originates from the proximal end of the posterior cerebral artery.

There are abundant anastomoses between the branches of the anterior choroidal artery and those distributed to the sole of the cerebral peduncle, so no infarction occurs.

(3) The cerebellum is manifested as: ① ipsilateral limb ataxia; ② cerebellar language; ③ a few patients may have nystagmus,

Dizziness, etc.

Currently, most people believe that when the superior cerebellar artery is occluded, it often manifests as cerebellar cortical infarction, and is rarely accompanied by brainstem involvement.