The formation of gallbladder crystals is closely related to people’s living and eating habits to a certain extent. The free calcium in the body will react with the oxalic acid in the food to form crystals. Gallbladder crystallization is mainly the precipitation of bile components. At this time, it has not yet had time to form large stones. If it is allowed to develop further, it will become gallstones. Therefore, the gallbladder crystals should be properly treated at this time to inhibit their development. So what is the best way to eliminate gallbladder crystals? Gallbladder crystals are formed when free calcium in the body combines with oxalic acid in the fruits and vegetables you eat, indicating that the body is calcium deficient. The thyroid gland extracts calcium from the bones to meet the blood's calcium demand, and the excess remains free in the body. Supplement calcium and magnesium tablets in time. Magnesium can prevent them from combining and the crystals will no longer increase. Also, eat a good breakfast and drink plenty of healthy water. The crystallization will gradually decrease or even disappear. It is best to use Nutrilite calcium magnesium tablets plus vitamins for better results. Gallbladder wall crystallization is caused by abnormal lipid metabolism in the body. Cholesterol is deposited in the submucosa of the gallbladder and is phagocytosed by macrophages, forming a lesion that protrudes diffusely or locally into the gallbladder cavity. Most of them have no specific manifestations and are often discovered accidentally during physical examination. It is a non-neoplastic benign lesion of the gallbladder. Cholesterol crystal deposition in the gallbladder wall is a type of gallbladder polyp, accounting for about half of the total number of gallbladder polyps. There are more men than women. After the gallbladder is opened, it can be divided into two types according to its appearance: (1) localized protrusion (polyp type); (2) diffuse flat type (strawberry type). These two types of lesions are often accompanied by varying degrees of chronic gallbladder inflammation. Most patients do not have any discomfort; a few patients may have mild upper abdominal distension, soreness in the right shoulder and back, and loss of appetite; some patients may experience upper abdominal colic (commonly known as biliary colic), acute cholecystitis, acute pancreatitis, bile duct bleeding, common bile duct obstruction (often jaundice) due to polyp detachment. So far, the formation mechanism of cholesterol crystals in the gallbladder wall is still unclear, so treatment methods are also being explored. When implementing medical treatment, in addition to a low-fat diet and moderate exercise, you should also dynamically observe whether the polyps are enlarging, whether their number is increasing, whether stones are formed, whether there is localized thickening of the gallbladder wall (which can be detected by B-ultrasound examination), and whether taking anti-inflammatory and choleretic drugs can relieve symptoms. The general treatment principle is: if accompanied by obvious symptoms, surgical treatment should be performed; if the symptoms are mild, dynamic observation should be performed, and anti-inflammatory and choleretic drugs can be taken intermittently if necessary. The indications for surgery are as follows: (1) combined with stones; (2) significant thickening of the gallbladder wall (greater than 0.4 cm); (3) accompanied by gallbladder emptying disorder (worsened upper abdominal distension and pain after meals, etc.); (4) complications such as acute cholecystitis, cholangitis, and common bile duct obstruction; (5) when tumor lesions cannot be ruled out during dynamic observation. |
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