Peripheral nerve pain?

Peripheral nerve pain during ejaculation is generally caused by disorders in the body's motor and autonomic nervous functions, which can manifest as numbness of the limbs, lack of strength, and pain. Peripheral nerve pain may also be a physical discomfort caused by peripheral neuritis. It is best to go to the hospital for relevant examinations for peripheral nerve pain, and take appropriate treatment measures based on the examination results. Peripheral nerve pain can generally be treated with important and nerve nourishing drugs, and you should also be careful not to overwork.

Symptoms of peripheral neuritis 1. Sensory disturbance:

In the early stage, the symptoms are often abnormal sensations such as burning, pain, numbness or hyperesthesia at the finger (or toe) tips, followed by gradual loss of sensation or even disappearance. The distribution of sensory disturbances is glove- or stocking-like. A small number of patients may have deep sensory impairment. There is often tenderness in the gastrocnemius muscle and other places.

2. Movement disorders:

The symptoms include decreased muscle strength, low muscle tone, weakened or absent tendon reflexes, and in some cases (such as furazolidone), the reflexes may be active. Muscle atrophy may occur after long-term illness.

3. Autonomic dysfunction:

The skin on the extremities is cool, pale, flushed or slightly cyanotic, with little or excessive sweating, the skin becomes dry, thin, tender or rough, and the nails lose their normal luster and have increased keratinization.

Examination of peripheral neuritis

Examination 1. Cerebrospinal fluid examination:

Normal or slightly elevated protein content.

Examination 2: Electromyography and nerve conduction velocity:

If there is only mild axonal degeneration, conduction velocity may be normal. When there is severe axonal degeneration and secondary demyelination, the conduction velocity slows down and the electromyogram shows abnormal neurological changes. When segmental demyelination occurs but axonal degeneration is not significant, the conduction velocity slows down, but the electromyogram may be normal. Measuring the electromyogram and nerve conduction velocity is helpful in distinguishing neurogenic damage from myogenic damage in this disease, and is also helpful in distinguishing axonal lesions from segmental demyelinating lesions. Axonal lesions are manifested as decreased amplitude, while demyelinating lesions are manifested as slower nerve conduction velocity.

Examination 3: Immunity examination:

For those suspected of having immune diseases, tests such as immunoglobulin, rheumatoid factor, antinuclear antibody, antiphospholipid antibody, as well as lymphocyte transformation test and rosette formation test can be performed.

Examination 4: Nerve biopsy:

Nerve tissue biopsy can provide more accurate evidence for determining the nature and extent of nerve damage.