What is the matter with the baby's high chest muscles

There are many reasons why a baby has high pectoral muscles. It may be caused by rickets. We know that rickets is a common disease in infants and young children. Its main cause is calcium deficiency in the body. If vitamin D is insufficient, it will also affect calcium absorption, which will cause adverse effects on the child's bone development. Once rickets occurs, it will cause great harm to the child's health.

Causes of Rickets

1. Perinatal vitamin D deficiency

Studies have shown that although daily supplementation of 400 IU of vitamin D by mothers in the late pregnancy has little effect on the 25-OH-D3 in the blood circulation of full-term infants, compared with mothers who regularly supplement during pregnancy, the vitamin D levels of newborns of mothers who do not supplement during pregnancy will quickly drop to a deficient level, so premature babies and twins are more prone to insufficient storage.

2. Insufficient sunlight

In urban life, tall buildings block sunlight; there is air pollution; the daylight is short and the ultraviolet rays are weak in the cold winter; there is not enough time for outdoor activities; or the skin is less exposed during outdoor activities; the climate, season, atmospheric cloud cover, latitude, skin color, and skin exposure can all affect the production of endogenous vitamin D.

3. Fast growth

Due to factors such as low weight, premature birth, twins, and illness, the baby will grow and develop relatively faster after recovery and need more vitamin D. However, the vitamin D stored in the body is insufficient, making rickets more likely to occur. 4. Insufficient vitamin D in food

Because natural foods contain little vitamin D, exclusive breastfeeding, and insufficient outdoor activities, the risk of developing rickets due to vitamin D deficiency increases if vitamin D is not supplemented.

5. Disease and drug effects

Gastrointestinal or hepatobiliary diseases affect the absorption of vitamin D, such as infantile hepatitis syndrome and chronic diarrhea. Severe damage to the liver and kidneys can lead to vitamin D hydroxylation disorders and insufficient production of 1,25-OH2-D3, causing rickets. Long-term use of anticonvulsant drugs can lead to vitamin D deficiency in the body. For example, phenytoin sodium and phenobarbital can stimulate the activity of the oxidase system in the microsomes of liver cells, thereby accelerating the decomposition of vitamin D and 25-OH-D3 into inactive metabolites. Glucocorticoids have the effect of antagonizing the transport of calcium by vitamin D.