TCM treatment of nystagmus

Many people have not heard of nystagmus. In fact, this is what people usually call eyelid twitching. This is mainly because the eyes do not get enough rest and are always in a relatively tired state, which leads to such symptoms. The best thing is to prescribe the right medicine, so that it can be improved to a certain extent, and it can also make your eyesight better.

1. Etiology treatment: symptomatic treatment.

2. Improve vision: For ocular nystagmus, the focus is to improve vision and prevent primary lesions that cause random vision. Appropriate prisms can also be prepared to eliminate compensatory head position and improve vision.

3. Surgical treatment: Those with congenital impulsive type (i.e. eye position): can undergo surgery, the purpose of which is to correct the compensatory head position according to the "middle zone" eye position, change the eye position, reduce nystagmus and improve vision. Because the group of extraocular muscles moving in the slow phase is strong, the group of extraocular muscles moving in the fast phase is weak. To this end, the two extraocular muscles on the slow phase side can be moved backward to reduce their tension and balance them with the group of extraocular muscles on the fast phase side. Move the "mid-band" eye position from an eccentric fixation position to a straight-ahead fixation position.

If the left side is a middle-zone static eye position nystagmus, the left lateral rectus muscle and the right medial rectus muscle can be moved back 6mm, the left medial rectus muscle can be shortened by 6mm, and the right lateral rectus muscle can be shortened by 7mm (generally the lateral rectus muscle is moved 2mm more back than the medial rectus muscle) to move the static eye forward.

3. Etiology and treatment: Nystagmus is not an independent disease, but a clinical manifestation. Therefore, we must first carry out symptomatic treatment according to the cause.

4. Surgical treatment can be used for congenital idiopathic nystagmus. Move the resting eye forward to improve vision and reduce or inhibit the occurrence of nystagmus. The method is to first determine the resting eye position, and then strengthen or weaken the medial and lateral rectus muscles of both eyes by 5, 6, 7 and 8 mm respectively, so that the resting eye is moved to the front.

1. Lesions of the vestibular organ of the inner ear are mostly horizontal nystagmus or mixed horizontal and rotational nystagmus, without vertical nystagmus. The amplitude of the nystagmus is small and the direction of the nystagmus is toward the healthy side, often accompanied by vertigo. It is common in inner ear vertigo (Meniere's disease), otitis media, labyrinthitis, acute vestibular function damage, pontocerebellar angle tumors, etc. Vestibular nystagmus is more persistent than inner ear nystagmus.

2. Medullary lesions are mostly rotational spontaneous nystagmus, and vertical nystagmus can occasionally be seen, which is seen in syringomyelia, vascular lesions, tumors, etc.

3. Pontine lesions often present as horizontal nystagmus, sometimes with abducens nerve palsy. Nystagmus can be seen atypically in tumors, vascular lesions, multiple sclerosis, etc.

4. Midbrain lesions are mostly severe orthostatic nystagmus, which is seen in vascular diseases, encephalitis, trauma, etc. If the lesion is near the oculomotor nucleus at the level of the superior colliculus of the midbrain, eyeball depression can be seen.

5. Cerebellar lesions often cause horizontal or rotational nystagmus. If the lesion occurs near the fourth ventricle, there is often a forced head position. Nystagmus may not be obvious when the cerebellar vermis is lesioned.