Can renal necrosis be cured?

We all know that renal necrosis is generally incurable. Once you suffer from renal necrosis, it almost means that the kidney is unusable. The only way to extend the service life of the kidney is through dialysis, or through kidney transplantation, a new kidney source is replaced to prolong the patient's life. However, renal necrosis can be divided into local necrosis and strong necrosis. Patients with local necrosis can be treated slowly. So can renal necrosis be cured?

Renal necrosis is generally unlikely to be cured, but localized necrosis can usually be compensated by normal renal units. You can go to a regular hospital or a tertiary-level A-class hospital's urology department for examination and treatment. Also pay attention to urine volume. If anuria occurs, dialysis treatment can be adopted. In addition, mentality is also very important. You must maintain a good mentality so that the condition will not worsen.

1. General edema: Almost all patients experience varying degrees of edema, which is most evident on the face, lower limbs, and scrotum. The swelling may persist for weeks or months, or may come and go throughout the course of the disease. After infection (especially streptococcal infection), edema often recurs or worsens, and even azotemia may occur.

2. Digestive tract symptoms: Due to gastrointestinal edema, there are often symptoms of digestive tract dysfunction such as loss of appetite, nausea, vomiting, abdominal distension, etc. The above symptoms are aggravated when there is azotemia.

3. Hypertension: It is an important clinical manifestation of non-nephrotic syndrome, but it is caused by water and sodium retention, increased blood volume, and temporary hypertension. Type II primary renal intercalary syndrome may be accompanied by hypertension.

4. Proteinuria: Large amounts of proteinuria are the most important condition for diagnosing this condition.

5. Hypoproteinemia: It is mainly characterized by a decrease in plasma protein, the degree of which is significantly related to the degree of proteinuria.

6. Hyperlipidemia: Triglycerides in the blood are significantly increased.

Renal necrosis, also known as acute renal failure, is a clinical syndrome in which the renal urinary function is sharply reduced due to the kidney itself or external factors, resulting in serious disturbance of the body's internal environment. The main manifestations are oliguria or anuria, azotemia, hyperkalemia and metabolic acidosis. Most people begin to experience oliguria (daily urine volume 50-400 ml) or anuria 12-24 hours after the precursor symptoms. It usually lasts 2-4 weeks. There may be anorexia, nausea, vomiting, diarrhea, hiccups, dizziness, headache, irritability, anemia, bleeding tendency, deep and rapid breathing, and even coma and convulsions. Accumulation of metabolites: increase in blood urea nitrogen, creatinine, etc. Metabolic acidosis occurs. Electrolyte imbalance: may include hyperkalemia, hyponatremia, hypermagnesemia, hyperphosphatemia, hypocalcemia, etc. Especially hyperkalemia. In severe cases, it can lead to cardiac arrest. Water imbalance can easily lead to excessive water retention; in severe cases, it can lead to heart failure, pulmonary edema or cerebral edema.