Right parietal lobe meningioma

We eat whole grains every day and face various external stimuli, which often make us prone to some diseases. We may feel unfamiliar with some diseases. For example, many people have never heard of the term right parietal lobe meningioma. In general, right parietal lobe meningioma is a benign tumor. When suffering from this disease, patients will experience some discomfort and should be treated under the doctor's advice.

1. Sensory impairment

Sensory disturbances are divided into general sensory disturbances and cortical sensory disturbances. General pain and temperature disturbances caused by parietal tumors are mostly not obvious. Even if they occur, they all occur at the distal end of the limbs and are very mild glove or sock-type sensory disturbances. This is because the thalamus also receives some pain and temperature impulses. Cortical sensory disturbances are mainly manifested as disturbances in the position sense, two-point discrimination, tactile positioning and pattern sense of the limbs on the opposite side of the lesion. For example, when the patient closes his eyes, he can feel the object held in his hand, but he cannot judge the weight, size, shape, texture, etc. of the object, and even cannot recognize simple numbers written on the skin. Therefore, he cannot complete a comprehensive analysis of the object, which is called loss of solid sense. This is the result of extensive damage to the superior parietal lobule of the postcentral gyrus. Cortical sensory disturbances can also manifest as sensory neglect. When the affected limb is stimulated, the sensation may be completely normal or slightly reduced. If both limbs are stimulated at the same time, only the healthy limb will feel it, and the affected limb will be completely ignored. When the factors of tactile stimulation of the affected limb have been removed, the patient still feels that the stimulation continues for a period of time, which is called tactile retention.

2. Body image disorder

Patients have difficulty recognizing their own structure. This phenomenon is particularly common in patients with lesions in the right parietal lobe. The mechanism of its occurrence has not been fully understood. There are many clinical manifestations. For example, patients are indifferent to their hemiplegia, do not pay attention to it, as if it has nothing to do with them, and have no anxiety. This is called hemiplegic agnosia. Patients completely deny their hemiplegia, and even when the paralyzed limbs are shown to them, they firmly deny that they are their own limbs. Sometimes they think they are someone else's hands or feet, or they use irrelevant reasons to explain the inability to move their limbs. This phenomenon is called hemiplegic agnosia. Some patients have a feeling of losing their limbs, and believe that their limbs no longer exist, that the paralyzed limbs are not their own, and that their limbs have been lost. Another manifestation is the feeling that one or more limbs are present. This manifestation is called phantom polylimbia. In addition, some patients also experience finger agnosia, left-right disorientation, and autoamnesia.

3. Amorphosis

Astructure is also called structural apraxia. It refers to the inability to correctly recognize and distinguish the structural arrangement of spatial objects, buildings, paintings, patterns, etc. that involve spatial relationships. The patient cannot combine them, cannot understand the relationship between them, and cannot use tools correctly to work, which makes life difficult. Clinically, it can be examined by drawing, building blocks, etc. Although the patient can imitate and the various components seem to be there, they lack the ability to layout and proportional relationships, or they are inverted upside down, left and right, the arrangement is too crowded or scattered, the original shape is lost, there is no concept of space, and there is a lack of three-dimensional relationship. There is no consensus on the mechanism of astructure.

4. Gerstmann syndrome

When lesions are found in the angular gyrus and supramarginal gyrus in the posterior and inferior part of the parietal lobe, as well as the part where the parietal lobe transitions to the occipital lobe, the clinical manifestations are mainly finger agnosia, left-right disorientation, agraphia, and acalculia. Finger agnosia is the most common and is often bilateral. When the patient is asked to show a specified finger, he or she cannot recognize the finger and is confused in the use of the finger, especially the thumb, little finger, and middle finger.