How to get anorexia

There are many causes of anorexia, which are closely related to psychological factors and some organic diseases. In modern society, many people hope to achieve body shaping by losing weight. At this time, they do not use some reasonable methods to keep fit, but use some excessive dieting methods. Excessive dieting is often an important cause of anorexia.

Causes of Anorexia

In the past, AN was often considered a disease closely associated with Western European and North American cultures; however, in recent years, with the development of globalization, the rapid development of the advertising industry, changes in eating habits, the emergence of a large fitness industry, and the transformation of women's social roles, there is increasing evidence that AN is also reported in many non-Western societies.

AN patients may have certain personality traits before the onset of the disease, such as low self-esteem, perfectionism, rigidity, conservatism, inflexibility, sensitivity, worry, rigor, introversion, timidity, hyperactivity, strong self-esteem, egocentrism, unsociability, childishness, fantasy, inability to stick to one's own opinions, indecision, etc., and have very high demands for success or achievement.

Genetic factors play a role in the onset of AN, which has been confirmed by family studies and twin studies. However, the inheritance pattern and gene loci of AN have not yet been established. In-depth research has been conducted on the neurobiology of AN, involving neurotransmitters such as 5-hydroxytryptamine (5-HT), norepinephrine (NE), dopamine (DA), etc. AN also has a variety of neuroendocrine abnormalities, and a variety of hormones or neuropeptides are related to appetite and satiety, and there are a variety of complex interactions between different hormones or neuropeptides;

For most neuroendocrine disorders, they are state-related and often return to normal after clinical recovery. In terms of brain imaging, several CT studies have shown that AN patients have enlarged CSF spaces (enlarged cerebral sulci and ventricles) when they are starved for a long time, and one study found that this condition recovered after weight gain. Functional imaging studies have found that AN patients have reduced metabolism and perfusion in the frontal and parietal cortices, and speculated that local 5-HT dysfunction occurs.

Prognosis: After a series of comprehensive treatments for AN, about 45% of patients have a good prognosis without any sequelae; about 30% of patients have a moderate prognosis, but still have many symptoms and body shape and weight problems; about 25% of patients have a poor prognosis, it is difficult to reach a normal weight, and the disease is chronic, recurring, and requires repeated hospitalization. 5% to 15% of patients eventually die from cardiac complications, multiple organ failure, secondary infection, suicide, etc. Patients with a short disease course and a young age of onset have a better prognosis.