Treatment of myocardial damage

There is a saying that there are always more solutions than problems, and the same is true in medicine. After generations of famous doctors’ in-depth research, many ways of treating diseases have been discovered, but they are still looking for more perfect methods. Modern medicine is developing rapidly, and I believe there will be more and more methods to treat diseases such as myocardial ischemia and myocardial damage. So today I will introduce to you the treatment methods for myocardial damage.

Soil, water and nutrition factors

According to investigation, this disease has obvious regional characteristics. The soil, water and food in the affected areas lack certain trace elements needed by the human body, such as selenium, molybdenum, magnesium or related nutrients, which interfere with myocardial metabolism, cause myocardial damage and thus become ill.

Except for post-pneumonia myocarditis or post-pneumonia heart failure, myocardial protection treatment must be intensified. Advice and suggestions: Patients are advised to rest in bed, supplement with vitamin C, eat a high-nutrition diet, and take intravenous fructose, coenzyme Q10 and other myocardial nutritional drugs.

Refers to markers whose blood levels increase within 6 hours after myocardial injury. Currently known early markers for diagnosing acute coronary syndrome (ACS) mostly appear in the early stages of the pathological process (before myocardial necrosis). The use of early markers can help with early diagnosis and, in turn, early treatment.

C-reactive protein (CRP) CRP increases abnormally in the early stage of myocardial operation and has a short window period. It has good clinical value in the early stage of myocardial injury and prognosis estimation. With the application of high-sensitive CRP (hs-CRP) detection method, its clinical application value has received increasing attention in recent years.

Myoglobin (Mb) Although Mb has low myocardial specificity, it can be rapidly released from the necrotic myocardium after myocardial infarction and has a high sensitivity. Mb has a short blood half-life, so it helps to observe whether reinfarction occurs during the course of AMI and whether the infarction expands. Mb is also a sensitive and accurate indicator for evaluating reperfusion during thrombolytic therapy of AMI.

Identify markers

A biochemical marker that appears in the blood 6 to 9 hours after the onset of myocardial injury and persists for several days, and has a high sensitivity and specificity for myocardial injury.

Creatine kinase isoenzyme (CK-MB) There are many methods for CK-MB analysis. The commonly used method to measure its activity (u/L) is immunosuppression analysis. The CK-MB mass analysis method is to measure its protein concentration (μg/L), which avoids interference (such as giant CK) that may be encountered in activity determination. It has high sensitivity (minimum detection limit <1μg) and accuracy, short measurement time (the fastest takes only 7 minutes), is suitable for automatic analysis, and has been widely recognized.

Cardiac troponin (cTn) cTn has two isoforms: cTnI and cTnT. Because of its high sensitivity, strong specificity, and long duration after onset, cTn is currently a better definite marker for diagnosing myocardial injury.