Can eating barley rock sugar prevent recurrence of condyloma?

Condyloma acuminatum is a common skin disease that occurs frequently in people. People who suffer from this disease need to pay attention to many issues, whether in diet or daily life. Genital warts are a contagious disease, and clothing or bed sheets in daily life may be sources of infection. Diet can also easily cause warts to worsen, so people with warts need to pay attention to their diet. But can eating barley rock sugar prevent recurrence of condyloma?

Traditional Chinese medicine believes that coix seed can remove dampness. Genital warts are a sexually transmitted disease caused by human papillomavirus (HPV) infection, with proliferative lesions in the anal and genital area as the main manifestation. It is a viral infection. It is unscientific to say that eating coix seed and rock sugar can prevent recurrence of condyloma acuminata.

What are the daily dietary matters after the occurrence of genital warts?

1. Regularly taking honey or royal jelly can help enhance your own resistance and immunity. The recurrence of genital warts often occurs when the resistance is low;

2. Dietary precautions for genital warts: After removing the warts, you should quit smoking and drinking. In addition, alcohol, chili peppers, onions, garlic, leeks, ginger, coriander, dog meat, and mutton are also not allowed;

3. Consume more foods rich in vitamins, such as apples, barley, ginger, grapes, straw mushrooms, onions, tea, peaches, broccoli, asparagus, white fungus, bitter melon, carrots, spinach, sweet potatoes, and aquatic shellfish, which can enhance the body's immune function. Yogurt, garlic, and mushrooms have antiviral effects and can stimulate immune function, so you should consume more of them. Yogurt can enhance the activity of natural killer cells and promote the body's ability to kill viruses.

4. Dietary precautions for genital warts: Eat less seafood, such as lobsters, crabs, freshwater carp, etc., and avoid alcohol and spicy foods;

5. Patients with condyloma acuminatum should eat more foods high in protein and do physical exercise at the same time;

So what are the causes of its disease?

1. Causes of disease

Human papillomavirus (HPV) belongs to the genus Papillomavirus A, a group of small DNA viruses with a diameter of 55nm, no lipoprotein envelope, and a symmetrical icosahedron composed of 72 viral capsid particles. Its genome is a circular double-stranded DNA containing 7.9kb and a molecular weight of 5000000. In the study of HPV gene structure and function, its open reading frames (ORFs) E6, E7, E1, E2, E4, E5, L1, L2 and E3, E8 and the corresponding gene-encoded proteins have received great attention. HPV has many types and subtypes. People initially identified a new HPV type by having a cross-hybridization rate of less than 50% with known HPV type DNA, and identified a new subtype by having a cross-hybridization rate of more than 50% but different endonucleases. The currently used typing standard is: if the E6, E7 and L1 sequences of the undetermined HPV genome ORF have a homology of less than 90% compared with the known types, it is a new type; if there is a difference of 2% to 10%, it is a new subtype; if the difference is less than 2%, it is a homotypic variation. Based on PCR technology, HPV has now been divided into more than 100 types, of which about 75 have completed molecular cloning and gene sequencing.

There are at least 35 types of HPV that can infect the urogenital tract epithelium. The clinical manifestations are type-specific, resulting in a wide spectrum of diseases ranging from subclinical infections detectable by nucleic acid technology, subclinical infections detectable by histocytology technology, genital warts, atypical hyperplasia, carcinoma in situ, Bowen's papulosis, squamous cell carcinoma, etc. HPV6 and HPV11 are highly correlated with genital warts, and others include HPV16, HPV18, HPV30, HPV31, HPV33, HPV42, HPV43, HPV54, HPV55, etc. A large amount of research data also proves that HPV16, HPV18, HPV31, HPV33, HPV35, HPV39, HPV45, HPV51, HPV52, HPV54, HPV56, HPV66, HPV68, etc. are related to the occurrence of genital epidermal tumors.

2. Pathogenesis

Genital warts HPV can easily infect the squamous epithelial cells of the mucous membranes and skin, and small wounds in the area of ​​sexual contact promote the occurrence of infection. The a6 integrin in the basal cell layer may be the receptor for virus attachment, and the L1 protein plays a coordinating role when the virus binds and enters the cell. HPV in basal cells has weak antigenicity and can easily escape recognition and elimination by the body's immune system. Its genes express E1 and E2 in the early stages. The E1 protein is a nucleic acid phosphorylating phospholipid protein with adenine and guanine triphosphorylase activity and DNA helicase activity; the E2 protein is both an activator and a limiter of transcription, initiating transcriptional regulation by fixing on the 12-nucleoside recovery compound (ACCN6GGT). As the cells differentiate into spinous cells, complete virus particles carrying high-copy (>50) HPV DNA appear in the middle and upper cells. The proteins encoded by E6 and E7 play an important role in transforming cells, especially in high-risk HPV (HPV16, HPV18) infections. In summary, the early region E1-E8 of ORF is mainly responsible for virus replication and has transformation characteristics, while the late regions L1 and L2 are related to proliferation and replication. Virus particles are assembled during the terminal differentiation of keratinocytes, and progeny viruses are released when dead corneocytes are shed.

In the study of humoral immunity, HPV antigens have been extracted from viral particles, bacterially expressed fusion proteins, synthetic peptides, etc. Most results show that host antibodies are mainly targeted at capsid protein L2. The latest gene recombinant capsomerin L1 or L1

Studies on L2 antigen (VLPs) have shown that HPV antibodies are type-specific, positive results are strongly correlated with disease history, and antibodies are produced very slowly and in low titers. In a group of women recently infected with HPV16, the average time for serum antibodies to turn positive was nearly one year, and the average titer was 1:100. The duration of serum antibodies is still unclear, but there are reports that genital warts-type antibodies can persist for decades. The cellular immune response to HPV has always been considered to play an important role in inhibiting viral reactivation and wart regression. The intensity of T cell response is related to the occurrence, duration and regression of warts. Recent studies have found that lymphoproliferative responses to E6 and E7 are associated with the regression of skin lesions and

It is related to the clearance of HPV infection; cytotoxic T lymphocytes (CTLs) corresponding to E4 protein have been detected in the peripheral blood, draining lymph nodes and cancer tissues of women with cervical cancer.

A large number of HPV vaccination animal experiments have shown that preventive vaccination with viruses or genetically recombinant capsid proteins L1, L2, E7, E6 or E5 can protect the host from viral attack in whole or in part, or at least accelerate the occurrence of rejection reactions. Currently, reports on Phase I and Phase II clinical trials of human HPV vaccines have been seen. People hope to identify effective therapeutic vaccines from genetically recombinant proteins such as E7 and E6 to expand the treatment of HPV-related diseases.

Interferon has antiviral, antiproliferative and immunomodulatory activities. It has become an auxiliary drug for the treatment of HPV infection, especially generalized and refractory genital warts, with an effective rate reported at 40% to 60%. Factors affecting efficacy include: type, dosage and course of treatment, route of administration, patient's cellular immune activity (related to skin lesions), and HPV type. Dose-related side effects limit the treatment of larger doses of systemic administration. Currently, intralesional injection therapy is mostly used, and there is no standardized protocol. Due to cost and inconvenience of administration, treatment is often limited to patients for whom other treatments have failed. Some authors believe that intralesional injection therapy has good early treatment effects and report that the efficacy of treating primary condyloma acuminatum (HPV6 or HPV11) is greater than 70%.

After the occurrence of genital warts, it is of course very important to actively carry out scientific treatment, but it is also indispensable to restore the body's resistance as soon as possible through a reasonable diet in daily life. As long as you can follow the above-mentioned dietary therapy matters, I believe that genital warts can be defeated.