What are the symptoms of sequelae of encephalitis? What is the reason?

Sequelae of encephalitis are a group of infectious diseases of the central nervous system with prominent manifestations of mental and consciousness disorders. So what are the symptoms of sequelae of encephalitis?

Symptom 1: Autonomic nervous system attack (diencephalic): may be headache type, abdominal pain type, limb pain type, syncope type or cardiovascular type.

Symptom 2: Generalized tonic-clonic seizure (grand mal seizure): sudden loss of consciousness, followed by tonic and then clonic spasms. The symptoms of post-encephalitis sequelae are often accompanied by screaming, cyanosis, urinary incontinence, tongue biting, white or bloody foam in the mouth, and dilated pupils. After lasting for tens of seconds or minutes, the convulsion stops naturally and the patient enters a coma. After waking up, there is a short period of dizziness, irritability, and fatigue, and the patient cannot recall the entire process of the attack. If the seizure continues and the patient remains in a coma, it is called a grand mal seizure, which is often life-threatening.

Symptom 3. Complex partial seizures (psychomotor seizures): psychosensory, psychomotor and mixed seizures. The symptoms of sequelae of encephalitis often include varying degrees of impaired consciousness and obvious thinking, perception, emotion and psychomotor disorders. There may be manifestations of automatism such as fugue and sleepwalking. Sometimes, under the control of hallucinations and delusions, violent behaviors such as hurting others and self-injury may occur.

Symptom 4. Absence seizure (petit mal seizure): sudden interruption of mental activity, loss of consciousness, and may be accompanied by myoclonus or automatism. Each attack lasts from a few seconds to more than ten seconds. The electroencephalogram shows a spike-slow or sharp-slow wave complex 3 times per second.

Experts remind: The root cause of the sequelae of encephalitis is essentially the degeneration of nerve cells, a decrease in cell number and a decline in function. Neuroscience theory once believed that neurons in the central nervous system stopped growing shortly after birth and had no ability to regenerate after being damaged. Traditional treatment methods are mainly based on decompressing the compressed spinal cord and using neurotrophic factors to promote recovery, but they are often powerless when the effect is not obvious.