Can smoke inhalation disease heal itself?

Can smoke inhalation disease heal itself?

Moyamoya disease is a relatively rare disease. Although its incidence rate is not particularly high, once the patient is infected, the harm is very serious. It often leads to cerebral hemorrhage or cerebral infarction, which may cause obvious disability and even death. Moyamoya disease cannot heal itself. Once the disease is discovered, it should be treated in time. There are many treatment methods, the most common of which is surgical treatment. Patients should pay more attention to health care in daily life.

Can smoke inhalation disease heal itself?

In fact, once you get smoke inhalation disease, generally speaking, it is impossible for it to heal on its own. If the smoke progresses, it will only get worse. Many patients come to ask, "My blood vessels are different from those of normal people. If I check again after a period of time, will they become good blood vessels?"

So far, there is no precedent for this, and it is impossible. As for moyamoya disease, it will only get worse. Moyamoya disease is a dynamic change and it is also divided into six stages. From the first issue, to the second, third, fourth, fifth, to the sixth issue. During these six stages, its blood vessels are constantly changing. But the blood vessels will never become the same as those of normal people.

Clinical manifestations 1. Transient ischemic attack (TIA) is the most common type, accounting for about 70% of all idiopathic moyamoya disease. The clinical characteristics are repeated transient paralysis or weakness, mostly hemiplegia, but can also be alternating hemiplegia or double hemiplegia. Motor function was fully restored after the attack. The course of the disease is usually benign, with a tendency to spontaneously resolve or completely cease attacks. Rarely, it is accompanied by hemiconvulsions, headaches or migraines. Rarely, transient sensory disturbances, involuntary movements, or intellectual impairment may occur. 2. Infarction-type acute stroke, leading to persistent paralysis, aphasia, visual impairment and intellectual disability. 3. Epileptic type: Frequent epileptic seizures, partial seizures or status epilepticus, accompanied by epileptic-like discharges on the electroencephalogram. 4. Hemorrhagic subarachnoid hemorrhage or intracerebral hemorrhage. The probability of this type occurring in adult patients is higher than that in children.

The last three types of the above clinical classification are collectively called "non-TIA type". The course of the disease is complex and changeable, the prognosis is poor, and it often manifests as a mixed type, such as epilepsy type plus infarction type, epilepsy type plus TIA type, etc. If it is a simple epileptic seizure, the prognosis may not be very poor. Regardless of the type, those who develop the disease before the age of 4 have a poor prognosis. In addition, clinical symptoms and their severity depend on the compensatory effect of collateral circulation. If adequate cerebral blood perfusion can be maintained, clinical symptoms may not occur, or there may be only brief TIA-type attacks or headaches. If cerebral blood perfusion cannot be maintained, symptoms will be severe and cause extensive brain damage.

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