Atrophic gastritis

Nowadays, the pace of life and work is very fast, which leads to many people having irregular diet. If this continues for a long time, more and more people will suffer from stomach problems. After suffering from stomach problems, patients will show pain and bloating in the stomach, and often experience belching and loss of appetite. Atrophic gastritis is a type of stomach disease. If you want to effectively prevent atrophic gastritis, you must know the causes of the changes.

Causes of atrophic gastritis 1. Helicobacter pylori (Hp) infection Hp can be cultured in the gastric mucosa of 60% to 90% of patients with chronic gastritis. In 1986, the 8th meeting of the World Society of Gastroenterology proposed that Hp infection is one of the important causes of chronic gastritis. 2. Eating habits: smoking, drinking, food stimulation, drugs that damage the gastric mucosa, etc.

3. Immune factors: In the blood, gastric juice or plasma cells of atrophic mucosa of patients with atrophic gastritis, especially gastric body gastritis, parietal cell antibodies or intrinsic factor antibodies can often be found. Therefore, autoimmune reaction is considered to be a related cause of atrophic gastritis. 4. Reflux of bile or duodenal fluid. 5. Physical factors Clinical statistical results show that the occurrence of this disease is significantly positively correlated with age. The older you are, the weaker the "resistance" of your gastric mucosa becomes, making it more susceptible to damage from adverse external factors. 6. The role of genetic factors in the onset of type A atrophic gastritis has been confirmed. The positive rates of PCA and IFA are high and atrophic gastritis is common in families with pernicious anemia. 7. Workers who come into contact with lead have a high incidence of gastric ulcers, and gastric mucosal biopsy found that the incidence of atrophic gastritis is also increased. In addition to lead, many heavy metals such as mercury, copper and zinc can cause certain damage to the gastric mucosa. 8. Radiation therapy for ulcers or other tumors can damage or even atrophy the gastric mucosa. 9. Iron deficiency anemia Many facts show that iron deficiency anemia is closely related to atrophic gastritis.

Treatment methods 1. General treatment: Quit smoking and avoid drinking, avoid using drugs that damage the gastric mucosa such as aspirin, indomethacin, erythromycin, etc., eat regularly, avoid overheated, salty and spicy foods, and actively treat chronic oral, nasal and pharyngeal infections. 2. Weak acid treatment: Patients with low or no acid as confirmed by pentagastrin test can take rice vinegar in appropriate amount, 1 to 2 spoons each time, 3 times a day; or 0.5 to 1.0 ml of 10% dilute hydrochloric acid, before or during meals, and take pepsin mixture at the same time, 10 ml each time, 3 times a day; multi-enzyme tablets or pancreatic enzyme tablets can also be used for treatment to improve indigestion symptoms. 3. When using anti-Helicobacter pylori to treat atrophic gastritis, gastric acid is reduced or lacking, and bacteria multiply in the stomach, especially Helicobacter pylori, which has a high detection rate. Anti-Hp treatment should be carried out. 4. Inhibit bile reflux and improve gastric motility . Cholestyramine can complex bile salts refluxed into the stomach and prevent bile acid from damaging the gastric mucosal barrier. Sucralfate can bind to bile acids and lysolecithin and can also be used to treat bile reflux. Ursodeoxycholic acid may also be given. Drugs such as metoclopramide, morphine, and cisapride can enhance gastric motility, promote gastric emptying, assist gastric and duodenal motility, prevent bile reflux, and regulate and restore gastrointestinal motility. 5. Increase mucosal nutrition . Albizzia julibrissin can increase gastric mucosal renewal, improve cell regeneration ability, enhance the resistance of gastric mucosa to gastric acid, and achieve the effect of protecting gastric mucosa. You can also use blood-activating agent; or sucralfate, urea capsules, carbenoxolone, prostaglandin E, etc.

<<: Low iodine

>>: Knee periosteum injury