Hyaline membrane disease

Hyaline membrane disease

The main cause of pulmonary hyaline membrane disease is the abnormal circulation of congenital lung tissue, which will produce some normal lung tissue through lung diseases. At the same time, it mainly affects the development of the lungs, congenital dysplasia and some suspected early lungs. In daily life, these diseases must be treated in time and simply prevented. The main causes in daily life are congenital pulmonary emphysema and some other types of lung diseases.

Therefore, there are many pathogenesis mechanisms in daily life. The main clinical manifestations are pain, weakened breathing, and inflammation of the trachea and bronchus. There are also many treatment methods in daily life.

1. Although congenital lobar emphysema is caused by congenital lung developmental disorder, there are also cases of acquired bronchial compression. Therefore, this disease is also called "neonatal" lobar emphysema or "infant" lobar emphysema, and it is not appropriate to call it "congenital" lobar emphysema. Its characteristics are: excessive expansion of one lobe or a section of lung tissue, compressing normal lung tissue, mediastinal organs and cardiovascular system. It is one of the common causes of acute respiratory distress in infants and young children. It is only seen in newborns or young children. One-third of cases develop the disease immediately after birth, 50% develop it within 1 month after birth, and only 5% develop it within 6 months after birth. It is more common in males than in females. It is common in the upper lobes of both lungs (the left upper lobe is the most common), followed by the right middle lobe, and rarely in the lower lobe. The clinical manifestations are unilobar or unilateral hyaline membrane disease. Theoretically, abnormalities in the lung parenchyma itself are a possible cause of this disease, but this has not been confirmed. Some people have studied the morphology of the lung lobes after resection and found that the number of alveoli increased, exceeding 50% of the normal number, and the size of the alveoli was normal or enlarged, and the number and structure of the trachea and blood vessels were normal, suggesting that this disease is alveolar cystomegaly or an abnormal increase in alveoli after birth. 2. Specific emphysema (Swyer-James syndrome) Swyer and James (1953) first reported this disease in a 6-year-old boy. In 1954, Macleod reported 9 similar adult patients, so this disease is also called Macleods syndrome or MSJ syndrome. Pathology: The main manifestations are chronic inflammatory changes, without bronchial stenosis or obstruction, which is different from congenital lobar emphysema; the pulmonary artery is developed and filled, but relatively small, which is different from the pulmonary artery not occurring or not developing.

The above are the common treatments for pulmonary pulmonary permeability disease. The main clinical manifestations are pain, weakened breathing, and inflammation of the trachea and bronchus. There are also many treatment methods in daily life. You can take some medicines. The treatment of the infection is not her precautions in life. She must keep a light diet, eat more fresh food, more fruits and vegetables, and eat less spicy and irritating food.

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