Will nephritis cause back pain? This treatment is effective

There are no sensory nerves in the renal parenchyma, so low back pain is not the main symptom of patients with nephritis. However, the renal capsule, renal pelvis and ureter are nerve-distributed, so pain in the renal area is related to traction and increased tension of the renal capsule and spasm or increased tension of the renal pelvis and ureter.

1. Will nephritis cause back pain?

There are no sensory nerves in the renal parenchyma, so low back pain is not the main symptom of nephritis patients. However, the renal capsule, renal pelvis and ureter are nerve-distributed, so renal pain is related to the traction and increased tension of the renal capsule and spasm or increased tension of the renal pelvis and ureter. Such as renal colic caused by kidney stones and ureteral stones, and chronic dull pain in the kidney area caused by chronic nephritis, pyelonephritis, and polycystic kidney disease. In addition, low back pain can also be caused by many other reasons, such as lumbar muscle strain, lumbar bone hyperplasia, lumbar twisting, women's pelvic lesions, etc. Therefore, if you have low back pain symptoms, you need to be examined to make a clear diagnosis.

2. What is nephritis?

The physiological functions of the kidneys are mainly to excrete metabolic products and regulate the balance of water, electrolytes and acid-base, secrete a variety of active substances, maintain the stability of the body's internal environment, and ensure the body's normal physiological functions. Nephritis is a group of kidney diseases that are immune-mediated and involve inflammatory mediators (such as complement, cytokines, reactive oxygen species, etc.), which ultimately lead to inflammatory changes in the kidney's intrinsic tissue and cause varying degrees of renal function impairment. It can be caused by a variety of causes. Non-immune, non-inflammatory mechanisms are also involved in the chronic process.

3. Treatment of nephritis

3.1 General treatment

This includes avoiding fatigue, eliminating triggers such as infection, avoiding contact with nephrotoxic drugs or poisons, adopting a healthy lifestyle (such as quitting smoking, exercising moderately, and controlling emotions), and a reasonable diet. During the acute phase, you should rest in bed and gradually increase your activity after the clinical symptoms improve. A low-salt diet (less than 3g per day) should be given during the acute phase. People with normal renal function do not need to restrict protein intake, but in cases of azotemia, protein intake should be restricted, with high-quality animal protein as the main source. People with oliguria should limit their fluid intake.

3.2 Treatment targeting etiology and pathogenesis

Treatment targeting the immune pathogenesis often includes glucocorticoids and immunosuppressants. Blood purification treatments such as plasma exchange and immunoadsorption can effectively remove autoantibodies and antigen-antibody complexes from the body. Treatment targeting non-immune pathogenesis, including hypertension, hyperlipidemia, hyperglycemia, hyperuricemia, obesity, proteinuria, renal hypercoagulability, renin-angiotensin system activation, and oxidative stress. Renin-angiotensin system blockers, such as ACEI/ARB, are one of the most important therapeutic measures to delay the progression of kidney disease.