Can I get pregnant if I have hyperthyroidism? Can patients with hyperthyroidism have children?

Can I get pregnant if I have hyperthyroidism? Can patients with hyperthyroidism have children?

Can you get pregnant if you have hyperthyroidism? Can patients with hyperthyroidism have children? Married women with hyperthyroidism should actively treat their hyperthyroidism. It is more appropriate to wait for hyperthyroidism to heal after pregnancy. Suggestions: Thioureas for the treatment of hyperthyroidism, including thiouracil, methylthiourea and propylthiouracil, can reach the fetus through the placenta. Excessive doses can cause fetal brain dysplasia and can cause fetal goiter and hypothyroidism. Giant thyroid gland can cause fetal labor and asphyxia. In addition, hyperthyroidism can aggravate the condition of patients with hyperthyroidism and even cause more serious complications.

Causes

Causes of hyperthyroidism include diffuse toxic goiter (also called Graves' disease), inflammatory hyperthyroidism (subacute thyroiditis, painless thyroiditis, postpartum thyroiditis, and Hashimoto's hyperthyroidism), drug-induced hyperthyroidism (levothyroxine sodium and iodine-induced hyperthyroidism), hCG-induced hyperthyroidism (transient hyperthyroidism due to vomiting of pregnancy), and pituitary TSH tumor hyperthyroidism.

Clinically, more than 80% of hyperthyroidism is caused by Graves' disease, which is an autoimmune disease of the thyroid gland. The patient's lymphocytes produce thyroid-stimulating immunoglobulin-TSI. Clinically, the TSI we measure is the thyrotropin receptor antibody: TRAb.

The cause of Graves' disease is currently unclear. It may be related to factors such as fever, lack of sleep, and mental stress, but clinically the cause of the disease cannot be found in the vast majority of patients. Graves' disease is often combined with other autoimmune diseases, such as vitiligo, alopecia, type 1 diabetes, etc.

Clinical manifestations

Thyroid hormones promote metabolism and the body's redox reactions. Hypermetabolism requires the body to increase food intake; gastrointestinal activity is enhanced, and the frequency of bowel movements increases; although food intake increases, the oxidation reaction is enhanced, the body's energy consumption increases, and patients experience weight loss; increased heat production manifests as heat intolerance and sweating, and some patients experience low fever; increased thyroid hormones stimulate sympathetic nerve excitement, and clinical manifestations include palpitations, tachycardia, insomnia, sensitivity to surrounding things, mood swings, and even anxiety.

If patients with hyperthyroidism do not receive proper treatment for a long time, they will suffer from weight loss and hyperthyroid heart disease. Patients who are emaciated are often more susceptible to acute infectious diseases, which can lead to disability or death. Hyperthyroidism heart disease causes heart enlargement, arrhythmia, atrial fibrillation and heart failure, causing patients to lose their ability to work and even die.

examine

Physical examination revealed an enlarged thyroid gland (mild to severe). In elderly patients, the enlargement is often not obvious and the thyroid gland is soft or medium in texture. In severe patients, vascular murmurs can be heard throughout the entire period using a stethoscope. In severe hyperthyroidism, tremors can even be felt when touched by hand. The heart rate of most patients with hyperthyroidism is increased, often exceeding 90 beats per minute when at rest, and elderly patients may exhibit rapid atrial fibrillation. Patients with hyperthyroidism have hot flashes on the skin and fine hand tremors. Many patients also have eyelid edema, widened palpebral fissures, less blinking in both eyes, and conjunctival congestion and edema. Severe patients may experience exophthalmos, restricted eye movement, and even incomplete eyelid closure.

Some patients with more severe hyperthyroidism experience myxedema in the anterior tibia (shinbone) of the lower limbs. The skin in front of the tibia becomes coarser, thicker, and rougher, with an orange peel-like appearance. The sweat hair becomes coarser, similar to elephantiasis, and is quite difficult to treat.

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