Diffuse thyroid disease

People cannot predict what disease will occur. But once it occurs, it should be treated promptly. The thyroid gland is the most common organ in the human body that suffers from disease, and there are many problems related to thyroid diseases. For example, diffuse thyroid disease is a serious disease that requires surgical treatment. However, if not treated in time, it can easily lead to disease. So what should we do if we have diffuse thyroid disease?

Diffuse thyroid disease is a disease of the thyroid gland. Clinically, common types of diffuse thyroid disease include nodular goiter (hereinafter referred to as nodular goiter), thyroid cancer (hereinafter referred to as thyroid cancer), Hashimoto's thyroiditis (hereinafter referred to as Hashimoto's disease), Graves' disease, etc. Because the treatment plans for each disease are different, it is necessary to

Qualitative diagnosis of such diseases before treatment.

prevention

1. Try to avoid head and neck X-rays during childhood.

2. Maintaining a happy spirit and preventing emotional injury are important aspects of preventing the occurrence of this disease.

3. In view of water and soil factors, pay attention to dietary adjustment, eat kelp, clams, seaweed regularly and use iodized salt. But too much iodine can be harmful, and in fact may be another risk factor for certain types of thyroid cancer.

4. Thyroid cancer patients should eat nutritious foods and fresh vegetables, and avoid fatty, dry, and spicy foods.

5. Avoid the use of estrogen, as it promotes the occurrence of thyroid cancer.

6. Thyroid proliferative diseases and benign tumors should be treated actively and regularly in the hospital.

7. After radiotherapy and chemotherapy after thyroid cancer surgery, actively using Chinese and Western medicine for preventive treatment is an effective way to improve the efficacy.

8. Exercise actively to improve disease resistance.

Apathetic hyperthyroidism

Apathetic hyperthyroidism is a special type of hyperthyroidism, which is more common in the elderly. The patient's laboratory tests meet the diagnostic criteria for hyperthyroidism, but are slightly elevated or at the upper limit of normal. Patients do not have typical clinical manifestations of hyperthyroidism, such as hyperactivity, heat intolerance, and sweating. On the contrary, the clinical characteristics of apathetic hyperthyroidism are: apathetic expression, depression, dullness, drowsiness, weight loss, generally no exophthalmos, the thyroid gland may be slightly enlarged or not enlarged, often with nodules, and a slightly increased heart rate. This type of hyperthyroidism is easy to be ignored due to its atypical symptoms, but this type is prone to crisis. Once it occurs, the patient will quickly enter a coma, so it deserves attention.

Subclinical hyperthyroidism

Subclinical hyperthyroidism refers to hyperthyroidism in which FT4 and FT3 levels are normal but TSH is below normal. Causes can be divided into temporary and persistent. The main temporary ones are: subacute thyroiditis, Hashimoto's thyroiditis, postpartum thyroiditis, drug-induced thyroiditis, etc. The main persistent ones include: Graves' disease, autonomous functional thyroid adenoma, etc. Subclinical hyperthyroidism may not have any symptoms and most cases do not require treatment, but those with high titers of thyroid autoantibodies are more likely to progress to clinical hyperthyroidism or clinical hypothyroidism and require further treatment. Subclinical hyperthyroidism can be divided into exogenous and endogenous types. The former mainly refers to that caused by drugs (including ultrasonic doses of thyroid hormone). In addition, thyroiditis caused by patients with multinodular goiter taking iodine can also manifest as subclinical hyperthyroidism. The latter refers to those caused by thyroid diseases (including Graves' disease, multinodular goiter, and autonomously functioning thyroid adenoma). The risk of osteoporosis may be increased in patients with subclinical hyperthyroidism, and subclinical hyperthyroidism may increase the risk of atrial fibrillation, and subclinical hyperthyroidism is one of the risk factors for atrial fibrillation. Aure et al. studied 23,628 subjects and found that the incidence of atrial fibrillation in those with serum TSH concentrations less than 0.14 milliunits/liter (Mu/L) was 13.3%, which was significantly different from that in those with normal serum TSH concentrations, with a relative risk of 5.2%. Appropriate treatment is recommended.