Convulsions and foaming at the mouth

Convulsions and foaming at the mouth
In the past, people had little research and understanding of carbuncle, so many people could not get good treatment and the pain attacks could not be controlled. But since the 1980s, science has developed rapidly, many new drugs have been introduced, and the level of pain treatment has been greatly improved. Facts have proved that, generally speaking, more than 80% of boils can be cured. The cure rate for pediatric patients is higher, and some types can be cured nearly 100%. This result is far more optimistic than people expected. Early and regular treatment is the main factor that determines the effectiveness of treatment. So, let’s understand what is going on when your whole body is twitching and foaming at the mouth in real life!

1. Introduction to the disease Due to the different starting sites and transmission methods of abnormal discharges, the clinical manifestations of epileptic seizures are complex and diverse, and can manifest as paroxysmal movement, sensory, autonomic nervous system, consciousness and mental disorders. There are many causes of epilepsy. After regular anti-epileptic drug treatment, the seizures of about 70% of epilepsy patients can be controlled, and 50% to 60% of them can be cured after 2 to 5 years of treatment. Patients can work and live like normal people.

2. The disease classification currently commonly used is the epilepsy seizure classification scheme proposed by the International League Against Epilepsy in 1981. Epileptic seizures are classified into partial/focal seizures, generalized seizures, and unclassifiable seizures. In 2010, the International League Against Epilepsy proposed the latest classification scheme for epileptic seizures, which reclassified and supplemented epileptic seizures. Although the new program summarizes the progress of epilepsy research in recent years, it is more comprehensive and complete. 1. Partial/focal seizures: refers to seizures in which the initial symptoms and EEG changes indicate that "neurons in a certain part of the cerebral hemisphere are activated first." Includes simple partial seizures, complex partial seizures, and secondary generalized seizures. 2. Generalized seizure: refers to a seizure in which the initial symptoms and EEG changes indicate that "both cerebral hemispheres are affected simultaneously." Includes absence, myoclonic, tonic, clonic, tonic-clonic, and atonic seizures. 3. Unclassifiable seizures: seizures that cannot be classified due to insufficient or incomplete data, or seizures that cannot be classified in the current classification standards (such as convulsive seizures). New types of seizures confirmed in recent years include absence myoclonus, negative myoclonus, eyelid myoclonus, and gelastic seizures.

3. Classification of epilepsy syndromes: According to the different causes of epilepsy, it can be divided into idiopathic epilepsy syndrome, symptomatic epilepsy syndrome and possible symptomatic epilepsy syndrome. The new scheme proposed by the International League Against Epilepsy in 2001 also defined or standardized some key terms, including reflex epilepsy syndrome, benign epilepsy syndrome, and epileptic encephalopathy. 1. Idiopathic epilepsy syndrome: a syndrome in which, in addition to epilepsy, there is no structural brain damage and other neurological symptoms and signs. The disease usually starts before puberty and has a good prognosis. 2. Symptomatic epilepsy syndrome: lesions or abnormalities of the central nervous system caused by various reasons, including abnormal brain structure or various factors affecting brain function. With the advancement of medicine and the continuous development and enrichment of examination methods, more and more epilepsy cases can find the causes. 3. Possible symptomatic epilepsy syndrome or cryptogenic epilepsy: It is considered to be a symptomatic epilepsy syndrome, but the cause is currently unknown. 4. Reflex epilepsy syndrome: refers to epilepsy in which almost all seizures are triggered by specific senses or complex cognitive activities, such as reading epilepsy, startle epilepsy, visual reflex epilepsy, hot bath epilepsy, card epilepsy, etc. If the triggering factors are removed, the attacks will disappear. 5. Benign epilepsy syndrome: refers to an epilepsy syndrome that is easy to treat or can be completely relieved without treatment and leaves no sequelae. 6. Epileptic encephalopathy: refers to the progressive brain dysfunction caused by epileptic abnormalities themselves. The main or entire cause is due to epileptic seizures or frequent epileptic discharges during the intervals between seizures. Most of the cases occur in newborns, infants and children. The EEG was obviously abnormal and the drug treatment was ineffective. Including West syndrome, LGS, LKS, Ohtawara syndrome, Dravet syndrome, etc. 4. Causes of epilepsy The causes of epilepsy are complex and diverse, including genetic factors, brain diseases, systemic or systemic diseases, etc. 1. Genetic factors Genetic factors are an important cause of epilepsy, especially idiopathic epilepsy. Molecular genetic studies have found that the molecular mechanism of some genetic epilepsy is structural or functional changes in ion channels or related molecules. 2. Brain diseases Congenital abnormalities of brain development: cerebral gray matter heterotopia, brain perforation malformation, tuberous sclerosis, cerebral facial angiomatosis, etc. Brain tumors: primary or metastatic tumors Intracranial infections: various encephalitis, meningitis, brain abscess, brain cysticercosis, brain toxoplasmosis, etc. Craniocerebral trauma: birth trauma, intracranial hematoma, cerebral contusion and various combined craniocerebral injuries, etc. Cerebrovascular diseases: cerebral hemorrhage, subarachnoid hemorrhage, cerebral infarction and cerebral aneurysm, cerebral arteriovenous malformation, etc. Degenerative diseases: Alzheimer's disease, multiple sclerosis, Pick's disease, etc.

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