What is the cause of the sunken blood vessels in the legs?

We are able to walk by relying on the strength of our legs. Without a pair of healthy legs, not only will our work be affected, but in serious cases we may not even be able to find a stable job. The legs contain muscles, bones, and various blood vessels. Leg blood vessel depression is a common leg disease among many people nowadays. So what exactly is going on with leg blood vessel depression?

It is considered to be caused by the depression of the epidermal veins. It can happen to normal people, so there is no need to be nervous. It has no major impact on your health, but you need to observe it. If there are local nodules, or redness and swelling, you need to see a doctor in time to rule out the possibility of thrombosis. Do moderate physical exercise, quit smoking and drinking, and eat more vegetables and fruits.

A thrombus is a small piece of blood formed when blood flows onto the surface of a peeling or repaired part of the inner surface of a blood vessel in the cardiovascular system. In the variable fluid-dependent form, the thrombus is composed of insoluble fibrin, deposited platelets, accumulated leukocytes, and entrapped erythrocytes.

Thrombosis is a multifactorial process in which a group of genetic and environmental factors interact and influence each other. The main characteristics of common thrombosis patients in clinical practice are familial inheritance, recurrent attacks, severe symptoms, abnormal sites of thrombosis formation, and young onset of disease.

Suspicion of arterial or venous thrombosis or thromboembolism requires objective evidence. Angiography is the standard of care, but ultrasonography, performed with skill, can also examine superficial vessels and the heart.

Among patients with spontaneous deep vein thrombosis documented by venography, a genetic predisposition is present in 25% to 50% of cases. The presence of a congenital defect in the anticoagulant mechanism (eg, factor V resistance to activated protein C, homocystinemia, protein C deficiency, protein S deficiency, antithrombin deficiency, fibrinolytic insufficiency) is sufficient to cause venous thromboembolism when combined with a thrombotic stimulus (eg, surgery, pregnancy, contraceptive use, antiphospholipid antibodies). Patients with multiple thrombosis in the early stage have a significantly higher frequency and severity of thrombosis than those with a single thrombosis.

Antithrombotic therapy is the use of thrombolytic drugs, antiplatelet drugs and anticoagulants. When developing an antithrombotic treatment strategy, the first thing to pay attention to is thrombolytic therapy, because thrombolytic drugs can remove an already formed thrombus. Antithrombotic treatment should be diversified, depending on whether the affected part is the venous or arterial circulation system; the extent and location of vascular involvement; the risk of thrombosis extension, embolism or recurrence; and the relative benefits and harms of antithrombotic treatment and bleeding.