How long does it take for oral lichen planus to heal?

Oral planus is a common skin disease, most of which are caused by fungal infections. Patients with oral planus should try not to eat seafood and spicy and greasy foods, and eat less onions, ginger, garlic, coriander, and toon sprouts, etc. These irritants will affect the surface healing time of ulcers. They should change their smoking and drinking habits and try not to eat irritating foods during the illness.

Diet and health care 1. What foods are best not to eat:

1) Avoid seafood.

2) Avoid spicy and irritating foods, such as chili peppers, pepper, alcohol, raw onions, ginger, garlic, etc.

3) Avoid irritating foods, such as beef, mutton, coriander, and toon. Irritants can delay the healing time of ulcer surface.

4) Quit smoking. Smoking during the onset of the disease may affect the healing of the ulcer surface.

1. What foods are good for your health?

1) As for the diet of patients with oral lichen planus, it is advisable to eat more fresh vegetables and fruits.

2) Patients with erosive ulcer should be given soft food.

Preventive care improves oral hygiene.

The pathological etiology is speculated to be caused by damage to epithelial basal cells mediated by autoreactive T lymphocytes.

There is currently no relevant content description for disease diagnosis.

Inspection method Laboratory examination:

There is currently no related content description.

Other auxiliary examinations:

Histopathology: epithelial hyperkeratosis or atrophy. Active basal cell degeneration, colloid bodies can be seen in the epithelium and nodules. The dermis may be infiltrated mainly by monocytes, and lymphocytes migrate into the epithelium, thus making the boundary between the epithelium and connective tissue unclear. Plasma cells are not the majority in biopsies of other skin lesions except in biopsies of gingival lesions. Sometimes, the basement membrane shows hyalinization, which is more obvious with PAS staining.

Immunohistochemical staining showed that most immunoglobulins were deposited in the colloid bodies and a few were deposited on the cell surface. Fibrin deposits were seen in the basement membrane area, the dermis was mainly infiltrated by CD4 lymphocytes, and the interepithelial and basal areas were mainly infiltrated by CD8 lymphocytes. Epithelial Langerhans cells are normal in number but are more pleomorphic and express HLA class II antigens. Oral lichen planus keratinocytes express HLA class II antigens, but it cannot be differentiated from nonspecific gingivitis, lupus erythematosus, and candidal cheilitis. The Langerhans cell antigenicity of drug-associated lichenoid eruption is different from that of idiopathic lichen planus, even though their clinical and histopathological manifestations are very similar.